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In addition, in both the acute phase and Long COVID, a single latent vector could be derived from these somatic and affective symptoms, demonstrating that these symptom profiles are the expression of a shared core, namely the acute COVID-19 and Long COVID neuropsychiatric symptoms [25, 26]

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This leads to abnormal immune regulatory mechanisms, which may activate pathogenic immune responses or invasive T cells with abnormal bacterial antigens and cause a loss of normal mucosal immune tolerance

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